@article{ART002196498},
author={KyoungWan YOON},
title={Apoptotic cell clearance and human diseases},
journal={CELLMED},
issn={3022-6805},
year={2017},
volume={7},
number={1},
pages={3-3}
TY - JOUR
AU - KyoungWan YOON
TI - Apoptotic cell clearance and human diseases
JO - CELLMED
PY - 2017
VL - 7
IS - 1
PB - Cellmed Orthocellular Medicine and Pharmaceutical Association
SP - 3
EP - 3
SN - 3022-6805
AB - The efficient removal of dead cells is an evolutionarily conserved process essential for homeostasis in multicellular organisms. The phagocytosis involves a series of steps that ultimately leads the detection of apoptotic cell by the phagocytes and the subsequent engulfment and degradation of corpse. The uptake of apoptotic cells by phagocytes not only removes debris from tissues but also generates an anti-inflammatory signal that blocks tissue inflammation. Conversely, impaired clearance of dead cells can cause loss of immune tolerance and the development of various inflammation-associated diseases such as autoimmunity, but can also affect cancer development. This review will discuss current understanding of the molecular mechanism of apoptotic cell phagocytosis and how they may be related to human diseases.
KW - apoptotic cell clearance;phagocytosis;inflammation;autoimmunity
DO -
UR -
ER -
KyoungWan YOON. (2017). Apoptotic cell clearance and human diseases. CELLMED, 7(1), 3-3.
KyoungWan YOON. 2017, "Apoptotic cell clearance and human diseases", CELLMED, vol.7, no.1 pp.3-3.
KyoungWan YOON "Apoptotic cell clearance and human diseases" CELLMED 7.1 pp.3-3 (2017) : 3.
KyoungWan YOON. Apoptotic cell clearance and human diseases. 2017; 7(1), 3-3.
KyoungWan YOON. "Apoptotic cell clearance and human diseases" CELLMED 7, no.1 (2017) : 3-3.
KyoungWan YOON. Apoptotic cell clearance and human diseases. CELLMED, 7(1), 3-3.
KyoungWan YOON. Apoptotic cell clearance and human diseases. CELLMED. 2017; 7(1) 3-3.
KyoungWan YOON. Apoptotic cell clearance and human diseases. 2017; 7(1), 3-3.
KyoungWan YOON. "Apoptotic cell clearance and human diseases" CELLMED 7, no.1 (2017) : 3-3.